Select publications:

Cao A, Li J, Asadi M, Basgen JM, Zhu B, Yi Z, Jiang S, Doke T, El Shamy O, Patel N, Cravedi P, Azeloglu EU, Campbell KN, Menon M, Coca S, Zhang W, Wang H, Zen K, Liu Z, Murphy B, He JC, D’Agati VD, Susztak K, Kaufman L. DACH1 protects podocytes from experimental diabetic injury and modulates PTIP-H3K4Me3 activity. J Clin Invest. 2021 May 17;131(10):141279. doi: 10.1172/JCI141279.(PMID: 33998601)

Zhu B, Cao A, Li J, Young J, Wong J, Ashraf S, Bierzynska A, Menon M, Hou S, Sawyers C, Campbell K, Saleem MA, He JC, Hildebrandt F, D’Agati VD, Peng W, Kaufman L. Disruption of the MAGI2-RapGEF2-Rap1 signaling axis contributes to podocyte dysfunction in congenital nephrotic syndrome caused by mutations in MAGI2. Kidney Int. 2019; 96(3):642-655. (PMID: 31171376)

Ni J, Bao S, Johnson R, Zhu B, Li J, Vadaparampil J, Smith CM, Campbell K, Grahammer F, Huber TB, He JC, D’Agati VD, Chan A, Kaufman L. MAGI-1 Interacts with Nephrin to Maintain Slit Diaphragm Structure Through Enhanced Rap1 Activation in Podocytes. J Biol Chem. 2016; 291(47): 24406-24417. (PMCID: PMC5114397)

Potla U, Ni J, Vadaparampil J, Yang G, Leventhal JS, Campbell KN, Chuang PY, Morozov A, He JC, D’Agati VD, Klotman PE, Kaufman L. Podocyte-specific Rap1GAP expression contributes to focal segmental glomerulosclerosis-associated glomerular injury. J Clin Invest. 2014; 124(4):1757-69. (PMCID: PMC3973092)