An abstract from the Hubmacher Lab was selected as a short talk for the ORS Tendon Section 2018 Conference “Discovery to Delivery in Tendon Research: Team Approaches”. Dirk will present the lab’s findings on the skeletal phenotype of a mouse model for geleophysic dysplasia in a talk titled: Absence of the extracellular matrix protein ADAMTSL2, mutated in geleophysic dysplasia, results in short tendons and reduced bone length.
