by hubmacherlab | Jun 27, 2019 | matrix biology, news, research
In a surprising twist, ADAMTSL2 gene expression was found in endothelial cells of the blood vessels in the forebrain (Supplemental Figure 6 in www.ahajournals.org/doi/full/10.1161/ATVBAHA.119.312388). However, it is absent in the vasculature outside of the brain. The...
by hubmacherlab | May 30, 2019 | conference, lab, news, research
Nandaraj’s abstract got selected for a podium presentation at the Gordon Research Seminar on Elastin, Elastic Fibers and Microfibrils. In addition, Stelios was chosen as a discussion leader for the keynote session.
by hubmacherlab | Feb 28, 2019 | matrix biology, news, publications, research
Geleophysic dysplasia is a short stature syndrome caused by mutations in ADAMTSL2 or FBN1. By deleting Adamtsl2 in the limb and in tendon, the Hubmacher lab developed a mouse model that has similar musculoskeletal features to patients with geleophysic dysplasia....
by hubmacherlab | Dec 20, 2018 | matrix biology, news, research, team, Uncategorized
The American Society for Matrix Biology interviewed Dr. Hubmacher for their most recent issue of “The Matrix Letter.” A copy of the newsletter can be found here: https://www.asmb.net/assets/docs/Newsletters/ML17-3.pdf
by hubmacherlab | Nov 14, 2018 | lab, news, team
We are pleased to welcome Sarah Stanley as a Master Student in the lab. She received her Bachelor of Science from the University of Massachusetts at Amherst and will be working on the role of ADAMTSL2 and other ECM proteins in skeletal muscle development. Welcome...
