by hubmacherlab | Sep 10, 2019 | lab, news, publications, research, team
Congratulations to Nandaraj and Stelios for their review article “The “other” 15–40%: The Role of Non‐Collagenous Extracellular Matrix Proteins and Minor Collagens in Tendon” published in a special edition of the Journal for Orthopedic Research. In addition, Dirk...
by hubmacherlab | Aug 22, 2019 | matrix biology, news, publications, research
We are very happy to share the final version of Limb- and tendon-specific Adamtsl2 deletion identifies a role for ADAMTSL2 in tendon growth in a mouse model for geleophysic dysplasia.
by hubmacherlab | Jun 27, 2019 | matrix biology, news, research
In a surprising twist, ADAMTSL2 gene expression was found in endothelial cells of the blood vessels in the forebrain (Supplemental Figure 6 in www.ahajournals.org/doi/full/10.1161/ATVBAHA.119.312388). However, it is absent in the vasculature outside of the brain. The...
by hubmacherlab | May 30, 2019 | conference, lab, news, research
Nandaraj’s abstract got selected for a podium presentation at the Gordon Research Seminar on Elastin, Elastic Fibers and Microfibrils. In addition, Stelios was chosen as a discussion leader for the keynote session.
by hubmacherlab | Feb 28, 2019 | matrix biology, news, publications, research
Geleophysic dysplasia is a short stature syndrome caused by mutations in ADAMTSL2 or FBN1. By deleting Adamtsl2 in the limb and in tendon, the Hubmacher lab developed a mouse model that has similar musculoskeletal features to patients with geleophysic dysplasia....
by hubmacherlab | Dec 20, 2018 | matrix biology, news, research, team, Uncategorized
The American Society for Matrix Biology interviewed Dr. Hubmacher for their most recent issue of “The Matrix Letter.” A copy of the newsletter can be found here: https://www.asmb.net/assets/docs/Newsletters/ML17-3.pdf
